The DIAMANT study reports hearing phenotypes encountered in patient with type-2 diabetes, with a particular focus on subclinical hearing loss associated with speech-in-noise intelligibility deficit.
A lightweight deep-learning pipeline classified chinchilla ABR waterfall plots to detect hidden cochlear pathology and differentiate synaptopathic-like changes from carboplatin-induced inner-hair-cell dysfunction.
Despite a permanent 40% loss of synapses, the auditory nerve achieves full functional recovery through a compensatory phenotypic shift that renders surviving fibers hyper-responsive, a mechanism that likely triggers central disorders such as tinnitus.
Extended high-frequency hearing sensitivity facilitates spoken-word recognition in listeners with normal audiograms.
This investigation objectively quantified speech-in-noise benefits obtained with the NAL-NL3 Minimal Hearing Loss (MHL) module for subclinical hearing loss using KEMAR and Ambisonic scenes, showing that occluding domes and advanced noise reduction features yield significantly greater SNR improvements than open domes and entry-level processing.
For a longitudinal cohort of 191 teenagers, physiological and behavioural metrics revealed no changes associated with recreational noise exposure over a three-year period, despite a wide range of exposures and instances of temporary threshold shift.
Behavioral and modeling results show that forward-masking estimates of cochlear gain reduction cannot be explained solely by MOC-mediated feedback in aging listeners.
The study demonstrates that poor sleep quality in young adults with normal hearing could be associated with elevated acoustic reflex thresholds with 1000 Hz probe tone
This study finds that the prevalence of tinnitus among school-aged adolescents in the southern region of India is 8.22%. The severity of tinnitus is notably affected by its frequency of occurrence and perceived loudness, but not by factors such as hearing thresholds, age, or gender.
This study uses efficient coding to explain how hidden hearing loss reorganizes adaptive auditory-midbrain representations in ways not captured by threshold audiometry or Fisher information alone.